Notch-HES1 signaling axis controls hemato-endothelial fate decisions of human embryonic and induced pluripotent stem cells
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Overview
authors
status
publication date
- August 15, 2013
has subject area
- 1102 Cardiorespiratory Medicine and Haematology (FoR)
- 1103 Clinical Sciences (FoR)
- 1114 Paediatrics and Reproductive Medicine (FoR)
- Apoptosis (MeSH)
- Basic Helix-Loop-Helix Transcription Factors (MeSH)
- Biomarkers (MeSH)
- Blotting, Western (MeSH)
- Cell Differentiation (MeSH)
- Cell Movement (MeSH)
- Cell Proliferation (MeSH)
- Cells, Cultured (MeSH)
- Dermis (MeSH)
- Embryonic Stem Cells (MeSH)
- Endothelium, Vascular (MeSH)
- Fibroblasts (MeSH)
- Flow Cytometry (MeSH)
- Gene Expression Profiling (MeSH)
- Gene Expression Regulation (MeSH)
- Hematopoiesis (MeSH)
- Hematopoietic Stem Cells (MeSH)
- Homeodomain Proteins (MeSH)
- Humans (MeSH)
- Immunoenzyme Techniques (MeSH)
- Immunology (Science Metrix)
- Induced Pluripotent Stem Cells (MeSH)
- Oligonucleotide Array Sequence Analysis (MeSH)
- RNA, Small Interfering (MeSH)
- Receptor, Notch1 (MeSH)
- Receptors, Notch (MeSH)
- Signal Transduction (MeSH)
- Transcription Factor HES-1 (MeSH)
published in
- Blood Journal