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GRP78 and CHOP modulate macrophage apoptosis and...
Journal article

GRP78 and CHOP modulate macrophage apoptosis and the development of bleomycin‐induced pulmonary fibrosis

Abstract

Endoplasmic reticulum (ER) stress and the unfolded protein response (UPR) have been associated with fibrotic lung disease, although exactly how they modulate this process remains unclear. Here we investigated the role of GRP78, the main UPR regulator, in an experimental model of lung injury and fibrosis. Grp78(+/-) , Chop(-/-) and wild type C57BL6/J mice were exposed to bleomycin by oropharyngeal intubation and lungs were examined at days 7 and …

Authors

Ayaub EA; Kolb PS; Mohammed-Ali Z; Tat V; Murphy J; Bellaye P-S; Shimbori C; Boivin FJ; Lai R; Lynn EG

Journal

The Journal of Pathology, Vol. 239, No. 4, pp. 411–425

Publisher

Wiley

Publication Date

August 2016

DOI

10.1002/path.4738

ISSN

0022-3417