Glucose transporter expression in L6 muscle cells: Regulation through insulin- and stress-activated pathways
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Overview
status
publication date
- July 1, 1997
has subject area
- 2,4-Dinitrophenol (MeSH)
- Animals (MeSH)
- Calcium-Calmodulin-Dependent Protein Kinases (MeSH)
- Cardiovascular System & Hematology (Science Metrix)
- Cell Differentiation (MeSH)
- Cell Line (MeSH)
- Enzyme Inhibitors (MeSH)
- Flavonoids (MeSH)
- Glucose Transporter Type 1 (MeSH)
- Glucose Transporter Type 3 (MeSH)
- Hypertonic Solutions (MeSH)
- Imidazoles (MeSH)
- Insulin (MeSH)
- Kinetics (MeSH)
- Mannitol (MeSH)
- Mice (MeSH)
- Mitogen-Activated Protein Kinase 1 (MeSH)
- Mitogen-Activated Protein Kinase 3 (MeSH)
- Mitogen-Activated Protein Kinases (MeSH)
- Models, Biological (MeSH)
- Monosaccharide Transport Proteins (MeSH)
- Muscle, Skeletal (MeSH)
- Nerve Tissue Proteins (MeSH)
- Phosphorylation (MeSH)
- Polyenes (MeSH)
- Protein Serine-Threonine Kinases (MeSH)
- Protein-Tyrosine Kinases (MeSH)
- Pyridines (MeSH)
- Ribosomal Protein S6 Kinases (MeSH)
- Signal Transduction (MeSH)
- Sirolimus (MeSH)
- Stress, Physiological (MeSH)
- Uncoupling Agents (MeSH)
- p38 Mitogen-Activated Protein Kinases (MeSH)
published in
- American Journal of Physiology Journal