What, How, and How Much Should Patients with Burns be Fed? Journal Articles

abstract

• The hypermetabolic response to severe burn injury is characterized by hyperdynamic circulation and profound metabolic, physiologic, catabolic, and immune system derangements. Failure to satisfy overwhelming energy and protein requirements after, and during, severe burn injury results in multiorgan dysfunction, increased susceptibility to infection, and death. Attenuation of the hypermetabolic response by various pharmacologic modalities is emerging as an essential component of the management of patients with severe burn injury. This review focuses on the more recent advances in therapeutic strategies to attenuate the hypermetabolic response and its postburn-associated insulin resistance.

authors

• Williams, Felicia N
• Branski, Ludwik K
• Jeschke, Marc
• Herndon, David N

status

• published 

publication date

• June 2011

has subject area

• 1103 Clinical Sciences (FoR)
• Animals (MeSH)
• Blood Glucose (MeSH)
• Burns (MeSH)
• Catecholamines (MeSH)
• Enteral Nutrition (MeSH)
• Humans (MeSH)
• Hypoglycemic Agents (MeSH)
• Insulin Resistance (MeSH)
• Lipolysis (MeSH)
• Liver (MeSH)
• Metformin (MeSH)
• Multiple Organ Failure (MeSH)
• Muscle, Skeletal (MeSH)
• Nutritional Support (MeSH)
• Parenteral Nutrition, Total (MeSH)
• Surgery (Science Metrix)
• Wound Healing (MeSH)

published in

• Surgical Clinics of North America  Journal

keywords

• Animals
• Blood Glucose
• Burns
• Catecholamines
• Enteral Nutrition
• Humans
• Hypoglycemic Agents
• Insulin Resistance
• Lipolysis
• Liver
• Metformin
• Multiple Organ Failure
• Muscle, Skeletal
• Nutritional Support
• Parenteral Nutrition, Total
• Wound Healing

Digital Object Identifier (DOI)

• 10.1016/j.suc.2011.03.002

start page

• 609

end page

• 629

volume

• 91

issue

• 3