Inhalation Toxicology
Journal
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Overview
publication venue for
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Letter concerning: Occupational exposures to cosmetic talc and risk of mesothelioma: an updated pooled cohort and statistical power analysis with consideration of latency period by Gary M. Marsh et al. (Inhal Toxicol. 2019 Aug 5:1–11. doi:10.1080/08958378.2019.1645768).
31:429-431.
2019
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Response to Marsh, G. M., Ierardi, A. M., Benson, S. M., & Finley, B. L. (2019). Occupational exposures to cosmetic talc and risk of mesothelioma: an updated pooled cohort and statistical power analysis with consideration of latency period. Inhalation toxicology, 31(6), 213–223.
31:385-386.
2019
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Re: Brent L. Finley, Stacey M. Benson & Gary M. Marsh (2017): Cosmetic talc as a risk factor for pleural mesothelioma: a weight of evidence evaluation of the epidemiology, Inhalation Toxicology, DOI: 10.1080/08958378.2017.1336187.
29:387-388.
2017
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Historical ambient airborne asbestos concentrations in the United States.
28:429-430.
2016
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Effect of diesel exhaust inhalation on blood markers of inflammation and neurotoxicity: a controlled, blinded crossover study.
28:145-153.
2016
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Pulmonary hypertension and vascular oxidative damage in cigarette smoke exposed eNOS−/−mice and human smokers.
24:732-740.
2012
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Letter re Marsh et al.
24:139-140.
2012
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Toxicogenomic Analysis of Mainstream Tobacco Smoke-Exposed Mice Reveals Repression of Plasminogen Activator Inhibitor-1 Gene in Heart.
21:78-85.
2009
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CYCLOOXYGENASE METABOLITES PLAY A DIFFERENT ROLE IN OZONE-INDUCED PULMONARY FUNCTION DECLINE IN ASTHMATICS COMPARED TO NORMALS.
12:1205-1224.
2000
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