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Transection of dysmyelinated optic nerve axons in...
Journal article

Transection of dysmyelinated optic nerve axons in adult rats lacking myelin basic protein

Abstract

Injury to myelin or oligodendrocytes may manifest as dysmyelinating or demyelinating conditions of the CNS. Previous studies using dysmyelinated animal models (myelin basic protein mutants) suggest possible axonal dysfunction with complete loss of myelin. In this present study, we evaluated retinal ganglion cell survival after axotomy in MBP mutants to determine if prolonged dysmyelination of CNS axons exerted a detrimental effect on neuronal …

Authors

Phokeo V; Ball AK

Journal

Neuroreport, Vol. 11, No. 15, pp. 3375–3379

Publisher

Wolters Kluwer

Publication Date

October 2000

DOI

10.1097/00001756-200010200-00022

ISSN

0959-4965