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Loss-of-function PCSK9 Mutants Evade the Unfolded...
Conference

Loss-of-function PCSK9 Mutants Evade the Unfolded Protein Response Sensor, GRP78, and Fail to Induce Endoplasmic Reticulum Stress when Retained

Authors

Lebeau P; Platko K; Al-Hashimi A; Byun JH; Lhotak S; Holzapfel N; Gyulay G; Igdoura SA; Cool DR; Trigatti B

Volume

32

Publisher

Elsevier

Publication Date

June 2018

DOI

10.1016/j.atherosclerosissup.2018.04.426

Conference proceedings

Atherosclerosis Plus

ISSN

2667-0909