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- Inhibition of ERK activation enhances the repair of double-stranded breaks via non-homologous end joining by increasing DNA-PKcs activation Academic Article
- Potent enantioselective inhibition of DNA-dependent protein kinase (DNA-PK) by atropisomeric chromenone derivatives Academic Article
- Unraveling the Complexities of DNA-Dependent Protein Kinase Autophosphorylation Academic Article
- XRCC4's interaction with XLF is required for coding (but not signal) end joining Academic Article