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BCL11A represses HBG transcription in K562 cells
Journal article

BCL11A represses HBG transcription in K562 cells

Abstract

BCL11A on chromosome 2p16 was recently shown to be a major quantitative trait locus for Hb F level and F-cell number in several populations with or without beta-hemoglobinopathy. We now show that BCL11A isoforms are expressed in K562 cells. Butyrate induction of HBG globin production in K562 is associated with reduced BCL11A. Conversely, augmented expression of BCL11A in K562 cells through transfection of BCL11A expression vector results in …

Authors

Chen Z; Luo H-Y; Steinberg MH; Chui DHK

Journal

Blood Cells Molecules and Diseases, Vol. 42, No. 2, pp. 144–149

Publisher

Elsevier

Publication Date

March 2009

DOI

10.1016/j.bcmd.2008.12.003

ISSN

1079-9796