Journal article
GM-CSF, IL-8, IL-1R, TNF-alpha R, and HLA-DR in nasal epithelial cells in allergic rhinitis.
Abstract
Epithelial cells potentially contribute to airways inflammation by antigen presentation and the production of proinflammatory cytokines. This study investigated the immunocytochemical localization of interleukin-8 (IL-8), granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukin-1 receptor (IL-1R Type I), tumor necrosis factor-alpha receptor (TNF-alpha R; 55kD), and human leukocyte antigen-DR (HLA-DR) on epithelial cells obtained …
Authors
Nonaka M; Nonaka R; Jordana M; Dolovich J
Journal
American Journal of Respiratory and Critical Care Medicine, Vol. 153, No. 5, pp. 1675–1681
Publisher
American Thoracic Society
Publication Date
5 1996
DOI
10.1164/ajrccm.153.5.8630619
ISSN
1073-449X
Associated Experts
Fields of Research (FoR)
Medical Subject Headings (MeSH)
AdolescentAdultEosinophilsEpitheliumGranulocyte-Macrophage Colony-Stimulating FactorHLA-DR AntigensHumansInterleukin-8KeratinsLeukocyte CountMacrophagesMiddle AgedNasal MucosaNeutrophilsReceptors, Interleukin-1Receptors, Tumor Necrosis FactorRhinitisRhinitis, Allergic, SeasonalT-Lymphocytes, Helper-InducerTumor Necrosis Factor-alpha