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Effect of an anti-Mo1 MAb on ozone-induced airway...
Journal article

Effect of an anti-Mo1 MAb on ozone-induced airway inflammation and airway hyperresponsiveness in dogs

Abstract

Ozone inhalation causes neutrophil migration into the airway and airway hyperresponsiveness in dogs. The leukocyte adhesion molecule Mo1 (CD11b/CD18) is a heterodimeric glycoprotein the expression of which is necessary for neutrophil adhesion to endothelium. To evaluate the contribution of Mo1 to ozone-induced neutrophil influx and airway hyperresponsiveness, six dogs were treated intravenously with an Anti-Mo1 monoclonal antibody (3.75 mg/kg …

Authors

Li Z; Daniel EE; Lane CG; Arnaout MA; O'Byrne PM

Journal

American Journal of Physiology, Vol. 263, No. 6, pp. l723–l726

Publisher

American Physiological Society

Publication Date

December 1, 1992

DOI

10.1152/ajplung.1992.263.6.l723

ISSN

0002-9513