Microtubule stability, Golgi organization, and transport flux require dystonin‐a2/MAP1B interaction

Loss of function of dystonin cytoskeletal linker proteins causes neurodegeneration in dystonia musculorum ( dt ) mutant mice. While much investigation has focused on understanding dt pathology, divergent functions of dystonin isoforms remain unclear. Here, we highlight novel functions of the dystonin‐a2 isoform in mediating microtubule (MT) stability, Golgi organization, and flux through the secretory pathway. Using dystonin mutant mice …